re.triple-g ghk-cu nad+ epithalon
the selection your protocol membership faqs explore compounds begin your protocol
belle labs · prefilled pen

epithalontime, reconsidered.

ala-glu-asp-gly · tetrapeptide · pineal bioregulator

your telomeres shorten every year.
your pineal gland produces less melatonin every decade.
your sleep architecture changes in ways that compound quietly.

epithalon addresses what most interventions don't reach.

€499
per penprefilled · pharmaceutical grade
request access →

available by request only

epithalon
what changes
the experience

your biology has
a clock. epithalon
speaks to it directly.

most describe it not as stimulation but as restoration. sleep that arrives differently. mornings that feel less like a negotiation. a quiet but unmistakable shift in how time feels on the body.

01
telomere lengthening
the only peptide with peer-reviewed evidence of telomerase activation in human somatic cells. the cellular clock responds.
02
sleep architecture
restores melatonin synthesis via pineal regulation — deep sleep stages return. the difference between sleep as a function and sleep as repair.
03
hormonal recalibration
cortisol rhythms normalise. the hypothalamic-pituitary axis resets. the hormonal shifts that accumulate with age begin to reverse their direction.
04
longevity signal
40+ years of russian institute research. lifespan extension in multiple models. the science behind epithalon is not speculative — it is documented.
4
amino acids
ala-glu-asp-gly. a tetrapeptide derived from the pineal gland. simple in structure. profound in effect.
40+
years of research
developed by professor vladimir khavinson at the st. petersburg institute of bioregulation. the most studied longevity peptide in existence.
coa
full documentation
certificate of analysis and batch traceability available for every order upon request.
for those who want to understand the science
+

epithalon (ala-glu-asp-gly) is a synthetic tetrapeptide developed from the pineal gland extract epithalamin, first isolated in the 1970s by professor vladimir khavinson at the st. petersburg institute of bioregulation and gerontology.

the primary mechanism involves activation of telomerase — the enzyme responsible for maintaining telomere length in somatic cells. published studies document measurable telomere elongation in human somatic cells following epithalon administration.

secondary mechanisms include normalisation of melatonin secretion via restoration of pineal gland activity, modulation of the hypothalamic-pituitary axis, and antioxidant effects documented across multiple tissue models. animal studies show consistent lifespan extension of 10–25% across multiple species.

for research contexts only. key references: khavinson et al. bull exp biol med 2003 · khavinson & morozov, ann n y acad sci 2003. available upon request.

belle labs · epithalon

the clock runs.
you decide the pace.

access is by request only.

€499 per pen

request access →