belle labs · prefilled pen

ghk-curestoration, visible.

glycyl-l-histidyl-l-lysine · copper complex · tripeptide

it occurs naturally in your plasma. it binds copper.
it signals your tissue to repair, rebuild, restore.
by 60, you have less than half the concentration you had at 20.

the body already knows how to regenerate. ghk-cu reminds it.

€499
per penprefilled · pharmaceutical grade
request access →

available by request only

Cu 2+ gly his lys ghk-cu
what changes
the experience

your tissue
remembers how
to repair itself.

ghk-cu doesn't create new biology. it restores the signalling your body has always had — the copper-bound tripeptide released at every site of injury, telling tissue to rebuild, collagen to form, inflammation to resolve.

01
skin & tissue restoration
stimulates collagen synthesis, tightens skin structure, and reverses thinning associated with aging. the extracellular matrix responds.
02
wound repair
clinical studies document accelerated wound closure, reduced neutrophil activity, and increased neovascularisation — tissue regeneration in measurable form.
03
antioxidant & anti-inflammatory
neutralises hydroxyl and peroxyl radicals. modulates nf-κb and nrf2 pathways — reducing oxidative stress at the cellular and molecular level.
04
gene expression reset
research suggests ghk-cu can reset elements of the genome — restoring gene activity patterns associated with younger, healthier tissue function.
3
amino acids
glycine, histidine, lysine. a tripeptide found naturally in human plasma — declining from 200 ng/ml at 20 to 80 ng/ml by age 60.
cu²⁺
copper complex
the copper binding is functional, not incidental. ghk-cu achieves maximum biological activity — neither component works the same alone.
coa
full documentation
certificate of analysis and batch traceability available for every order upon request.
for those who want to understand the science
+

ghk-cu (glycyl-l-histidyl-l-lysine copper²⁺) is a naturally occurring tripeptide found in human plasma, saliva, and urine. first identified in the 1970s, it has been the subject of extensive research for its roles in wound healing, tissue remodelling, and gene regulation.

at the molecular level, ghk-cu stimulates synthesis of collagen, elastin, decorin, and tissue inhibitors of metalloproteinases. it also modulates the nf-κb inflammatory pathway and activates nrf2-mediated antioxidant defences.

a clinical study in diabetic patients documented 98.5% wound closure in the ghk-cu group versus 60.8% in controls. gene expression research suggests ghk-cu can influence over 4,000 human genes — many associated with tissue repair, stem cell activation, and metabolic restoration.

for research contexts only. key references: pickart & margolina, int j mol sci 2018 · maquart et al. febs lett 1988. available upon request.

belle labs · ghk-cu

restoration
visible.

access is by request only.

€499 per pen

request access →